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Genetically engineered mice lacking NMDARs show deficits in habituation, working memory and associative learning (370)

Statistical analysis was rigorously designed to assess treatment efficacy and account for missing or incomplete data

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When these affinity-enhancing mutations were added to full-length rat amylin, all three mutated rat amylin analogs showed enhanced potency for amylin receptor 1/2 activation by 5- to 10-fold compared to endogenous rat amylin and a clinical drug pramlintide (Figure 9A, 9B and Table 3)

These effects may be optimally stimulated at lower concentrations where the body interprets the signal as a gentle prompt rather than an emergency