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BPC-157 upregulates survival genes (Akt1, KRAS, Src, Foxo1), reduces inflammatory mediators (IL-6, TNF-, NOS2, NFB), scavenges reactive oxygen species through its four carboxylic groups, activates vascular bypass pathways, and maintains blood flow despite vessel occlusion

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Insulin resistance correlates in most cases where there is a vitamin D deficiency, as it has been shown to be capable of modulating the homeostasis between glucose and insulin due to the action it exerts on the VDR

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[48] we observed increased oxidation in the presence of PUFA-plasmalogen PE (P-16:0/20:4)