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More broadly, genetic mouse models of AD have shown only limited success in predicting clinical outcomes, underscoring the need for caution when extrapolating preclinical efficacy to human disease (Syvanen et al., 2009

Furthermore, alanine mutations of the established glutathione-scaffold site residue D90 or of crucial residue K26 in monothiol PfGrx C32S/C88S slowed down the reaction and resulted in linear secondary diagrams for the first rate constant (Fig

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