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Regarding indel variants, we modeled the affected proteins and compared them with wild-type counterparts

doi: 10.1007/s00294-003-0415-2 32 DenegaI.d'EnfertC.Bachellier-BassiS

Osteoarthritis and Cartilage, 33 (8), 933-950

Under pathological conditions, oocyte development may be disturbed, resulting in follicular atresia and ovulatory dysfunction [103]

Specifically, HMGB1 released from necrotic cells binds and activates RAGEs, TLR-2, and TLR-4 which upregulate the expression of matrix metalloproteinase 9 (MMP-9), tumor necrosis factor- (TNF-), and interleukin (IL)-1, all of which promote cellular death and neuroinflammation [26]