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Such association can be attributed due to poor detoxification of RAN/tobacco toxins and lowering c-Jun phosphor-ylation due to its strong binding to JNK which consequently may inhibit apoptosis

Meanwhile, the level of ACSL4 (a common positive regulator of ferroptosis) was upregulated at the first hour after ph-IOP injury (Fig

Lung-on-chip also revealed to be an ideal platform for infectious diseases study, where the complex and dynamic device microenvironment generates essential data not achievable with classic 2D in vitro models (130, 131)