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Neuroglial metabolism in the awake rat brain: CO2 fixation increases with brain activity

Global public health burden of heart failure

Studies with mutants and transgenic plants over- or under-expressing enzymes or metabolites of the AsA-GSH pathway have proved very well the co-relation between this pathway and stress tolerance (Gill and Tuteja, 2010)

The abnormal proliferation of FLS results from a loss of contact inhibition that plays a critical role in RA by producing inflammatory cytokines and proteinases, such as matrix metalloproteinases (MMPs) and tissue inhibitors of metalloproteinases (TIMPs) that perpetuate joint destruction

Skeletal muscle protein synthesis is increased when mTORC1 is activated