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These surviving cells relied heavily on oxidative phosphorylation and were sensitive to oligomycin treatment, providing evidence that inhibition of mitochondrial function may effectively target cells that survive after suppression of oncogenic KRAS signaling [59]

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Other than this, UCB activates both extrinsic and intrinsic pathways of apoptosis as reflected by markers such as CD95, caspase-8, Bax, MMP, cytoplasmic Ca 2+ , caspase-3, and DNA fragmentation (143)