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Oxidative stress is implicated in the pathogenesis of IC/BPS as reactive oxygen species impair bladder function via their involvement in multiple molecular mechanisms

Most notably, several reports have highlighted how mNAM can increase H 2 O 2 production, either through Complex I inhibition [97], or by acting as a substrate for aldehyde oxidase enzymes [98]

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And as their name would apply, one would go, "Oh, they're activated by heat." Well correct, they are activated, very robustly, by heat and we can talk about that

Others have identified the role of SIRT-1 and -Klotho in mediating the anti-apoptotic actions of GABA in the -cells (47, 54)