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Dopamine release is mediated by voltage-gated calcium channels that trigger exocytosis of dopamine-filled vesicles into the synaptic cleft, where dopamine binds to its target receptors

Less consistent evidence across analyses were found for an association between higher C3 levels and an anhedonic symptom profile previously linked with inflammation and neurobiological reward processes

How close are we to complete annotation of metabolomes

doi: 10.1126/science.1170944 36 YalazaCAkHCagliMSOzgirayEAtaySAydinHHet al

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