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In normal somatic promoters, methylation of CGIs can result in gene silencing, either directly by impeding the binding of transcription factors or indirectly through the interaction of 5-methylcytosine (5mC) with methyl-CpG binding domain (MBD) proteins [25]

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DCS-treated patients tended to improve more, and more rapidly, though the difference did not reach statistical significance (Storch et al., 2010b)