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Additionally, contrasting with clinical results, homocysteine would inhibit both von Willebrand factor and thrombomodulin expression in cultured endothelial cells by affecting the redox-thiol status-regulated transport of newly synthesized glycoproteins in the endoplasmic reticulum

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All average concentrations were higher than those in the plasma

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Research in animal models shows combined use produces 30% faster healing with enhanced fibroblast activity and immune cell migration