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Thus, alterations in redox homeostasis may secondarily shape inflammatory signalling and cellular survival rather than reflecting a fixed pro- or anti-inflammatory role of KP metabolites

3.2.3 GPR56 G protein-coupled receptor 56 (GPR56) (ADGRG1) overexpression in CRC correlates with poor survival and increased postoperative relapse, with particular enrichment in microsatellite stable (MSS) disease-the predominant immune checkpoint inhibitor (ICI)-refractory subtype (90, 91)