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In addition to its role in redox buffering, GSH is a regulator of redox-dependent signaling and post-translational modification

In brief, a suspension of 5 ml (2.5 mg/ml) CB was sonicated in endotoxin-free water for 16 min and used at the same concentration as the i.t

Spurious group differences due to head motion in a diffusion MRI study

Expression of the Apx gene family during leaf senescence of Arabidopsis thaliana

The classic mechanism of acetaminophen hepatotoxicity is that its reactive metabolite, N-acetyl-p-benzoquinoneimine (NAPQI), depletes GSH and then covalently binds to proteins