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Furthermore, phosphorylated JNK induces oxidative stress, mitochondrial membrane permeability, and dysfunction through the activation of Bax, the translocation of Bax to the mitochondria, the opening of the mitochondrial permeability transition pore, and Src-dependent inhibition of the electron transport complex

ROS is considered a major physiological inducer of cellular senescence (Sun D

LFQ proteomics also revealed an absence of the gliotoxin oxidoreductase GliT in egtA 26933 (Supplementary Table S6)