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While ROS scavenging is beneficial in chronic diseases like diabetes (Zhang et al., 2020), excessive antioxidant supplementation may disrupt redox homeostasis, as evidenced by increased cancer risk in vitamin E trials (Poprac et al., 2017)

found activation of the polyol pathway and an upregulation of PARP-1 in the lungs of diabetic rats compared with nondiabetic animals, which correlated with increases in the NADH/NAD + ratio, the activities of mitochondrial respiratory complex IIV, and cellular ROS production (104), supporting the view that reductive stress occurs in the development of diabetic complications

The where, the when, and the how of immune monitoring for cancer immunotherapies in the era of checkpoint inhibition

Exposure to heavy metals, pesticides, and other harmful substances can lead to increased oxidative stress, necessitating a higher demand for antioxidants, including glutathione

Chemical drugs (such as cyclopamine and digoxin) (219, 220)