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Fatty liver disease, also known as hepatic steatosis, is a condition characterized by the accumulation of excess fat in liver cells

To maintain amino acid nutrition for tumor growth, the expression of the LAT1 ( SLC7A5 ) transporter is controlled by the pro-carcinogenic transcription factors c-Myc (Yue et al., 2017), HIF2 (Elorza et al., 2012), and NOTCH (Grzes et al., 2017), as well as the post-transcriptional regulator miR-126 (Miko et al., 2011)

Hence, several iPSC models for vasculopathy have also been described [37, 74, 83]

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