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have linked ALC to the bioenergetic abnormalities in SZ, suggesting ALC as a potential area for further study in the context of SZ [37]

XW, TM, YZ, PS and DQ provided scientific and technical assistance to the experiments, and critically revised the article for important intellectual content

Angiotensin Ii-Induced Vascular Dysfunction Depends on Interferon-- Driven Immune Cell Recruitment and Mutual Activation of Monocytes and Nk-Cells

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In addition, TMAO may accelerate the process of atherosclerosis and increase the risk of cardiovascular diseases through mechanisms such as promoting inflammation, increasing the expression of scavenger receptors, inhibiting reverse cholesterol transport, and enhancing vascular calcification (Luo et al., 2024)