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Given the protective effects of physiological levels of H 2 S against systemic inflammation, we hypothesized that CSE deficiency aggravates GalN/LPS-induced liver injury in mice

Therefore, we hypothesized that the antimicrobial effects of GSH might result from the overproduction of intracellular ROS

First, we confirmed that Cth mRNA expression is eliminated in the gastric tissues of Cth / mice with and without H

Many Firmicutes bacteria, including Bacillus and Staphylococcus species utilize bacillithiol (BSH) and cysteine as major thiol-redox buffers (Newton et al., 2009)

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