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found Escin-activated ROS to upregulate p38 expression in a dose- and time-dependent manner and induced apoptosis and autophagy in human osteosarcoma cells, but had a minimal impact on JNK and ERK-2 [28]

J Endocrinol, 198: 61-70

However, this type of autophagy can also consume excessive metabolic proteins, such as hexokinase 2 (HK2), impairing normal cellular metabolism and triggering unexpected cell death (163)