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Administration of 3-hydroxyanthranilic acid (3-HAA), a downstream tryptophan metabolite, ameliorated the symptoms of EAE by increasing the number of Treg cells and inhibiting the functions of Th1/Th17 cells (82)

This is expected pharmacology, not an unexpected side effect

The effects are so hard on the body

The peptide treatment mitigated bleomycin-induced transcriptional alterations in genes involved in stress response, inflammation, and fluid homeostasis, leading to a significant reduction in interstitial edema and down-regulation of proinflammatory and profibrotic factors

Figure 2 (B) Case map on three priority levels (I priority stands for critically high level