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While toll-like receptors (TLRs) signaling ensures protective immune response by recognizing invading pathogens and tissue-derived endogenous molecules, its overactivation perturbs the immune homeostasis by sustained release of pro-inflammatory mediators and subsequently underlies the development of many inflammatory diseases [296]

13,50,52 In this study, we found that Art significantly upregulated nuclear ATF3, which repressed SLC7A11 transcription and promoted ferroptosis in HSCs, and the results warrant further investigation

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Results : 240 patients were included in this study (median age 42 y, 84% male, 88% Hispanic, MELD-Na 27, IQR 22-31), and 12% (n=28) were prescribed MAT (TABLE)