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To examine the importance of this process, we took advantage of the potent GR inhibitor 2-AAPA (46), commonly used at 25 M (47)

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In summary, based on the characteristic metabolic reprogramming of tumor cells, combination therapies that simultaneously target multiple metabolic pathways or synergize with other treatment modalities represent a systematic strategy to overcome the limitations of monotherapy, suppress tumor adaptation, and ultimately increase therapeutic efficacy

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