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c-MET based targeted therapy c-MET has a major contribution towards compensating for inhibition of RTK pathways that help in proliferation and metastasis in HNSCC

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The effects of nanoparticles, microplastics, pharmaceuticals, and endocrine disruptors, and their interplay with climate-related physicochemical variation, have been identified as a research gap

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Gut microbiota-derived antigens are transported to the thymus via migratory dendritic cells, where they facilitate the expansion of antigen-specific T cells [59]