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ROS produced by radiation may be the original trigger for inducing ferroptosis in RILI, and the Keap1-NRF2 pathway has a protective effect against radiation-induced ferroptosis in alveolar epithelial cells (117)

GHK-Cu functions as a potent modulator of cellular antioxidant capacity, upregulating genes in the glutathione biosynthesis pathway and enhancing superoxide dismutase expression in fibroblast and keratinocyte model systems

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This step is crucial for the inflaming response, which now enables the maturation and release of IL-1 [47]

Mu H, MD, PhD, Konopleva M, MD, PhD, Jacamo R, PhD, Carter BZ, PhD, McQueen T, MS, Andreeff M , MD, PhD