Stefan BurAGomesALTeijeiroACampos-OlivasRMegasDBurSet alRegulation of OGT by URI in response to glucose confers c-MYC-dependent survival mechanisms
nh hng n ngi li xe v vn hnh my mc Khng nn s dng thuc khi li xe hay vn hnh my mc do thuc c th gy tc ng khng tt, nh hng ti qu trnh vn hnh my mc, li xe
This may result from tumor-mediated glucose restriction, which limits T-cell substrate availability and suppresses mTOR activity, glycolysis, and IFN- production, thereby promoting functional exhaustion while promoting tumor progression [171]
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Logistic issues: Extremely expensive (e.g., ~$25,000/treatment)
