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Reactive oxygen species (ROS) generated during catechol oxidation amplify DNA damage, while proliferative signaling from 16-OH increases mutational risk

This deposition of complement on an antigen, or on the antibody itself bound to the antigen, can flag the complex for engulfment by phagocytes with complement receptors on their cell surface

Meyer-Schwesinger C

Cytoskeleton and endopeptidase inhibitor activity emerged as the most abundant terms in the CCs and MFs, respectively

Guttiferone K isolated from Yunnan Baiyao was able to inhibit the downstream NF-B and Akt/mTOR signaling pathways via the TLR/IRAK-1 pathway, thereby significantly inducing autophagy and suppressing MTB-induced inflammatory responses in macrophages (113)