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Keywords: GHK peptide, fibroblasts, myofibroblasts, senescence, fibrosis, idiopathic pulmonary fibrosis The loss of cellular homeostasis in fibrosis is characterized by the accumulation of activated fibroblasts, known as myofibroblasts [1], which are responsible for excessive extracellular matrix deposition and tissue remodeling

We have identified changes in key antioxidant enzymes that may shed light on the mechanism of the protective effects of ALA

Currently, different treatment options are available, as shown in Figure 1C, including pharmacological treatment, but every treatment strategy has its own limiting factors

J Pineal Res 60(4):405414 Shi CS, Nabar NR, Huang NN, Kehrl JH (2019) SARS-coronavirus open reading frame-8b triggers intracellular stress pathways and activates NLRP3 inflammasomes