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Description

Of interest, treatment of aged rats with IGF-I was shown to upregulate eNOS and improve bioavailability of NO[295], and in vitro experiments in cultured endothelial cells demonstrated that IGF-I treatment reduce ROS production and upregulate eNOS[294]

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Description A dual-peptide compound studied across somatotropic, pituitary, and growth hormone secretagogue signaling pathways

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