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The increased metabolic activity also helps the body process nutrients more effectively, supporting both fat reduction and muscle recovery

Plant Signaling & Behavior 7, 16211633

10.1152/jn.00363.2021 J

A proposed mechanism of these beneficial outcomes is the inhibition of endoplasmic reticulum stress-induced apoptosis through extracellular signal-regulated kinase (ERK1/2) and the phosphoinositide 3-kinase (PI3K)/protein kinase B (Akt) signaling pathway in combination with the inhibition of adenylyl cyclase activity via Gi of the m2AChR, thereby reducing cAMP production and further attenuation of ER-stress and apoptosis (Fig

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