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In addition, extracellular TrxR1/Trx were related to cell signaling (Lveillard and At-Ali, 2017), inflammation and tumor progression (Sderberg et al., 2000)

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Hyperinsulinemia, insulin resistance, hyperglycemia and lipotoxicity all contribute to the creation of advanced glycation end products (AGEs), elevated free fatty acids and oxLDL, and can contribute to endothelial dysfunction, macrophage foam cell formation, inflammation and phenotypic switching of VSMCs, thereby supporting atherosclerosis (Zhao et al., 2024)