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This anorexigenic effect primarily resulted from a decline in the frequency of meals, even as the size of individual meals remained unchanged compared to that in the control group (21)

Senescent fibroblasts in diabetic wounds exhibit ferroptosis resistance due to defective ferritinophagy, but upregulating the expression of the ferritin autophagy-related molecule NCOA4 increases their susceptibility to ferroptosis and improves wound healing [81]

PMC 4753969

Notably, while markers of type I collagen synthesis and degradation are frequently used to indicate changes in bone turnover, they may also reflect changes in liver fibrogenesis or fibrinolysis (324, 325)

Furthermore, obesity has become an inducement to cause other diseases, and increases the risk of diabetes, NAFLD, and NASH under the stimulation of the chronic inflammation as well [98, 99]