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2000;12 Suppl 4:S74-9

During the bodys normal functioning an excess of oxygen containing molecules are produced, these molecules are typically very reactive with other molecules they come in contact with

The intensity of this inflammatory response is predictive of DCI and adverse outcomes, prompting a growing focus on the cellular and molecular mechanisms underlying aSAH induced neuro-inflammation

This process consists of ROS that appear to damage cellular membrane polyunsaturated fatty acids, resulting in the formation of toxic lipid peroxidation compounds [11]