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Dysregulated mitochondrial function, including increased reactive oxygen species production, disrupted intracellular calcium signaling, and impaired ATP synthesis, contributes to PD pathogenesis 9,10,11

In the MPTP mouse model, NRF2 activation has been associated with a reduction in oxidative stress (Williamson et al., 2012), induction of antioxidant enzymes (Jazwa et al., 2011), and improvement of the mitochondrial respiratory rate (Fu et al., 2018)

2026-02-21

The blood ammonia levels in children with GDH-HI will not increase significantly due to protein intake and are not related to blood glucose levels as well [12]

Involvement of oxidative stress in placental dysfunction, the pathophysiology of fetal death and pregnancy disorders