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Riboflavin supplementation to patients with multiple sclerosis does not improve disability status nor is riboflavin supplementation correlated to homocysteine

However, unlike DEM which depleted GSH in a specific manner, these agents interact with sulfhydryls from proteins, leading to impairment of vital cell functions

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Antioxidant N-Acetyl-L-Cysteine Ameliorates Symptoms of Premature Aging Associated with the Deficiency of the Circadian Protein BMAL1

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