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Toxic mechanisms of five heavy metals: mercury, lead, chromium, cadmium, and arsenic

PINK1-mediated Drp1S616 phosphorylation modulates synaptic development and plasticity via promoting mitochondrial fission

Therefore, we recommend that when deciding on the use of high-dose antineoplastic combination therapy to prolong survival of patients having persistent and resistant GBM tumors, the long-term effects of this treatment on cross-resistance and MDR phenotype should be analyzed in primary cell cultures obtained from patients

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We performed crystallographic analysis to further understand the structural and regulatory features of Hpy GS, which cannot be classified as either GS I- or GS I-