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Bridging the gap between translational and outcome research in cardiovascular disease

In this review, our analysis concentrates primarily on the UA-induced activation of immune cells, the consequent cytokine expression, and detrimental inflammatory responses arising from the imbalance in the immune cell populations that contribute to the progression of HUA and its comorbidities

Mottet NK, Vahter ME, Charleston JS (1997) Metabolism of methylmercury in the brain and its toxicological significance

This multidimensional, target-specific strategy holds promise for enhancing therapeutic efficacy while minimizing systemic side effects.Future large-scale longitudinal randomized controlled trials should prioritize structural and functional endpointssuch as TEWL, AMP expression, and tissue remodeling markerswhile incorporating refined stratification variables, including VDR polymorphisms, metabolic-nutritional status, and biological aging indices, to identify responsive subpopulations, optimize dosage windows, and enhance intervention efficacy

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