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Similarly, high carbohydrate consumption increases hepatic lipogenesis, thereby supporting fatty liver progression by increasing the synthesis and uptake of FFA [33, 34]

Resistin and visfatin levels are also significantly elevated and are independently associated with an increased risk for HS [159]

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Experimental limitations associated with direct in vivo measurements of ROS (detection limit for ROS using quantitative EPR spin trapping is ~ nM), which typically have short half-lives (seconds to s), require their quantification through indirect methods, such as DNA damage, membrane lipid peroxidation, and protein modifications