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However, mitochondrial dysfunction may also lead to numerous indirect negative consequences related to depleted levels of NADPH, which results from the distorted activity of this organelle [470,471,472]

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These changes induced ferroptosis, mitochondrial damage, and neuroinflammation in BV2 cells

This dysregulation involve increased ROS production, altered antioxidant enzyme levels, and disruption of mitochondrial function

The privately held company first filed for approval of the drug in September 1999 but the New Drug Application was rejected by the FDA in 2002