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As regulatory agents, organic selenium compounds can influence the expression of oncogenes and induce programmed cell death in cancer cells while also affecting cellular immune functions

10.1111/cns.13548 CNS Neurosci

These variants subsequently cause a RAS-protein gain of function associated with enhanced GTP activity, since GTPase-activating proteins (GAPs) are unable to bind KRAS, thus increasing the intrinsic GTPase activity of KRAS (Johnson et al