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NMN administration reduced damaged lysosomes and improved autophagy, thereby reducing heart failure and extending the lifespan in p32cKO mice

Chronic dysregulation of these UPR pathways leads to enhanced cardiomyocyte apoptosis, inflammation, and structural deterioration, forming a key mechanism underlying ER dysfunction in diabetic cardiomyopathy ( 6.2.2 Ca 2+ leakage The ER functions as the main intracellular calcium reservoir

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Acknowledgments We gratefully acknowledge the assistance of Home for Researchers for language modification

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