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Current evidence tends to suggest that mitochondrial transfer is primarily associated with downstream functional consequences of mtDNA mutations, such as mitochondrial dysfunction and metabolic stress, rather than being simply determined by specific mtDNA mutational sites per se [128, 370, 371]

Franco I, Viswanathan AN

Organoids: Stem Cells, Structure, and Function

,[21] is more stable in blood plasma than GSH, and enters the cells directly, where it is converted to reduced glutathione by the abundant cytoplasm thioesterases