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Although the primary behavioral outcomes of addictive substances are realized through distinct effector mechanisms, such as neurotransmitter transporters, ion channels, and receptor proteins, the common feature of these drugs is concerned with increased dopamine neurotransmission within the mesocorticolimbic circuitry of the brain from the ventral tegmental area to the nucleus accumbens and prefrontal cortex [1]

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it is likely that disease manifestation occurs when multiple risk factors coalesce

De Francesco EM, Vella V, Belfiore A