Exposure to these infectious pathogens and inflammatory stimuli can profoundly affect the brain and behavior ( Toxoplasma gondii infection, which, in animal models, alters behavior and increases the release of neurotransmitters such as dopamine, and the amount of dopamine release is correlated with the number of infected cells ( + T cells, which are crucial for sustaining lasting immunity, have been found to be downregulated in schizophrenia patients ( Toxoplasma gondii infection has been shown to affect the gut microbiome in mice ( Table 1 Clinical evidence for the association between infectious pathogens and schizophrenia
Purification and characterization of an ascorbate peroxidase from potato tuber mitochondria
Additionally, the advantages and disadvantages of these strategies are also assessed
Toxicol Sci 115(1):8997 Maes M et al (2011) Somatization, but not depression, is characterized by disorders in the tryptophan catabolite (TRYCAT) pathway, indicating increased indoleamine 2, 3-dioxygenase and lowered kynurenine aminotransferase activity
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