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Future work will use longitudinal profiling and single-cell sequencing to assess cell-type-specific GSH roles 5 Conclusion In conclusion, by leveraging multi-omics integration, the present study identified GSH metabolism as a pivotal node in SAE, while providing a comprehensive molecular framework for SAE pathogenesis, and linking microglia-driven neuroinflammation to hippocampal oxidative damage

Used BPC-157 400mcg plus Thymosin Beta-4 3mg twice weekly for 8 weeks post-operatively

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Laboratory Preparation: Reconstitute using an appropriate laboratory-grade diluent according to experimental protocol requirements

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