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One of the key regulators of oxidative stress in diabetes is thioredoxin-interacting protein (TXNIP), which acts as an endogenous inhibitor of the antioxidant thioredoxin (TRX)

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This adaptation is mediated by upregulation of the thioredoxin antioxidant system and an increase in surface thiol group density, forming a protective reducing barrier that shields NK cells from oxidative damage [176]