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Nitrated HGF ( bar 3 , peroxynitrite-treated at 1:2000) lost its activation activity as demonstrated by a drop in the activation index down to the baseline level observed in the negative-control culture ( bar 1 ) 32 and in GSSSG/LASSS-cultures ( bars 6 and 10 , missing HGF)

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Traditional dogma asserts that GST functions to catalyze glutathione (GSH)-substrate conjunction to preserve the macromolecule upon exposure to oxidative stress, thus defending cells against various toxic compounds

Many treatments have been devised, encompassing neurorestorative, anti-inflammatory, and neuroprotective agents

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