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A similar situation was observed in rbohD mutants of Arabidopsis, also exhibiting decreased apoplastic localization ( Figure 2 )

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Its enzymatic function is based on two aspects: its catalytic activity and the auto- S -glutathionylation of GST by itself on Cys47 and Cys10, both of which disturb the subsequent interaction with c-Jun NH 2 -terminal kinase (JNK), resulting in the formation of a GST multimer 30 (Figure 3)

Lipidomics analysis unravels aberrant lipid species and pathways induced by zinc oxide nanoparticles in kidney cells