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During signal transduction, the JAZ repressor protein, as a negative regulator, affects the transduction of JA signals by interacting with the MYC2 transcription factor [82]

Although tracing strategies based on membrane proteins like RAGE or CD31 are theoretically feasible, in the real-world scenario of ICU emergency care, existing immunoaffinity technologies often take too long and are too costly to rapidly enrich rare exosome subsets of specific origins (such as alveolar epithelial origin) with high purity from complex plasma components (138)