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Their formation is most prominent in mitochondria, where components of the electron transport chain, specifically complex I and complex III, occasionally leak electrons that react with molecular oxygen to produce superoxide anion

Preclinical investigations illustrate that its bioactive constituents may elevate monoamine concentrations, mitigate stress-induced neuroinflammatory responses, and stimulate neurotrophic factors such as BDNF, thereby facilitating neuronal survival and plasticity

Anti-inflammatory repair: inhibits the NF-B pathway and reduces the release of IL-6 induced by ultraviolet rays

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