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In this study, we investigated the mechanism underlying the protective role of melatonin against Cd-toxicity in two mammalian cell lines including WPMY-1 human prostate stromal cells and mouse embryonic fibroblasts (MEFs)

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Serum lipopolysaccharide-binding protein in endotoxemic patients with inflammatory bowel disease

Next, a metabolic-syndrome study reported higher energy expenditure, increased fatty-acid oxidation, reduced fat-mass accumulation, and improved insulin sensitivity in mouse models